What is the cause of central serous Chorioretinopathy?
What is the cause of central serous Chorioretinopathy?
Central serous chorioretinopathy is associated with increased sympathetic activity, and obstructive sleep apnea is known to cause such increases. Yavas and colleagues showed in a prospective study that 61% of patients with CSCR had underling obstructive sleep apnea diagnosed with overnight polysomnography.
What is serous fluid in eye?
Central serous chorioretinopathy, commonly referred to as CSC, is a condition in which fluid accumulates under the retina, causing a serous (fluid-filled) detachment and vision loss. CSC most often occurs in young and middle-aged adults. For unknown reasons, men develop this condition more commonly than women.
Can high blood pressure cause central serous retinopathy?
Some studies have shown that stress and high blood pressure may also increase the likelihood of developing central serous retinopathy.
What is the best treatment for CSR?
In CSR there is ischemic congestion of the choriocapillaris, and therefore a treatment focused at this level is needed. PDT is the best choice to achieve this. Inoue and colleagues7 recently used indocyanine green angiography (ICGA) to investigate which patients with CSR respond best to PDT treatment.
Can CSR be cured?
Treatment isn’t usually needed for CSR. Most people will find that their vision will improve within 3-6 months without the need for treatment. In a small number of people, CSR can be chronic, lasting longer than 12 months.
How common is CSR eye?
In CSR the macula becomes separated from the eye tissue behind it, and fluid builds up in the space created. It is six times more common in men than women, and most often affects people aged between 20 and 50. It normally affects only one eye, and up to half of people who get it once will have it again.
How is serous retinal detachment treated?
In typical central serous chorioretinopathy (CSC), serous retinal detachment usually resolves spontaneously with the return of visual function. If it persists, laser treatment to focal leaks outside the foveolar area is effective in resolving the detachment.
What causes PED?
The pathogenesis of PED formation in AMD is not completely understood, but it is believed to be due to the growth of choroidal neovascularization through Bruch’s membrane into the sub-RPE space with secondary extravasation of fluid or blood.
What does serous PED look like on Oct?
In cross-sectional OCT scans, serous PED appears as an optically empty dome-shaped elevation of the external high reflective band – the RPE, that steeply detaches from Bruch’s membrane (26). The overlying retina, usually adherent to the bullous PED, at the margins of the lesion]
How do you know it’s the temporal side of the Oct?
You know it’s the temporal side because the nasal side of the OCT has a thicker retinal nerve fiber layer (RNFL). Here is a BRVO with central macular edema. Chronic RVOs lead to inner retinal atrophy, which is also characteristic of the disease. Central serous chorioretinopathy (CSR)
Does central serous chorioretinopathy affect only one eye?
Central serous chorioretinopathy usually affects just one eye at a time, but both eyes can be affected at the same time. Central serous chorioretinopathy symptoms Symptoms of central serous chorioretinopathy can include: distorted, dimmed, or blurred central vision. a dark area in your central vision.
What is central serous chorioretinopathy (CSR)?
Central serous chorioretinopathy (CSR) CSR has a central SRF collection, no intraretinal fluid (IRF), and a thickened choroid. With CSR, there can often be a component of pigment epithelial detachment (PED) inside the area of serous detachment. These PEDs can be quite large.